A Fatal Adverse Effect of Barbiturate Coma Therapy: Dyskalemia
نویسندگان
چکیده
منابع مشابه
A Fatal Adverse Effect of Barbiturate Coma Therapy: Dyskalemia
The management guideline for traumatic brain injury (TBI) recommends high-dose barbiturate therapy to control increased intracranial pressure refractory to other therapeutic options. High-dose barbiturate therapy, however, may cause many severe side effects; the commonly recognized ones include hypotension, immunosuppression, hepatic dysfunction, renal dysfunction, and prolonged decrease of cor...
متن کاملProlonged Coma in Barbiturate Poisoning
In 1954 the recorded deaths from barbiturate poisoning in England and ^ were 574 and non-fatal cases were assessed as 6,000 (Locket, 1956). In spite of large number of reported cases in the literature, there were few in which coma1 prolonged for more than a week. The Medical Journal of Australia (1934) menti"' a case of ten days' duration, McElligott (1955) cited another of ten days, Shulman^ (...
متن کاملBarbiturate Coma: Rebound and Refractory Hyperkalemia
Purpose: To describe a traumatic brain injury patient who experienced profound dyskalemia upon the initiation and cessation of a pentobarbital infusion and propose management options for future patients receiving this intervention. Methods: Case report. Results: Case report. Conclusions: Dyskalemia has become an anticipated side effect of high dose barbiturate infusions in the setting of elevat...
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چکیده ندارد.
15 صفحه اولLife-Threatening Dyskalaemia after Barbiturate Coma Therapy: The Strategy of Management.
Barbiturate coma therapy (BCT) is a treatment option that is used for refractory intracranial hypertension after all other options have been exhausted. Although BCT is a brain protection treatment, it also has several side effects such as hypotension, hepatic dysfunction, renal dysfunction, respiratory complications and electrolyte imbalances. One less concerning but potentially life-threatenin...
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ژورنال
عنوان ژورنال: Korean Journal of Neurotrauma
سال: 2016
ISSN: 2234-8999,2288-2243
DOI: 10.13004/kjnt.2016.12.2.156